A20 was originally characterized as a TNF-inducible gene in human umbilical vein endothelial cells. As an inhibitor of NF-kappaB signaling, A20 could protect apoptosis, inflammatory and cardiac hypertrophy. We investigated the role of A20 on acute myocardial infarction (MI), ox-LDL-induced apoptosis in macrophage, and vascular remodeling. (1)We investigated the role of constitutive human A....