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Research progress of the dysfunction in foam cell emigration from plaques
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1.Department of Cardiology, ;2.Department of Pathology, Affiliated Hospital of Jiangsu University, Zhenjiang, Jiangsu 212001, China)

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R363

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    Abstract:

    The formation and inhibited emigration of foam cells are the key events in malignant remodeling of artery plaques. Advanced glycation end products (AGE) play an important role in the course of diabetes-accelerating atherosclerotic progression. Based on the latest international research progress and our existing results, this paper elaborated the mechanism of CD36 modulating foam cell emigration from plaques inhibited by Nε-carboxymethyl-Lysine, which would provide a new starting point for the treatment strategy of targeting foam cell migration mechanism in the future.

    Reference
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WANG Zhong-Qun, LI Li-Hua, YAN Jin-Chuan, YE Fei, SHAO Chen. Research progress of the dysfunction in foam cell emigration from plaques[J]. Editorial Office of Chinese Journal of Arteriosclerosis,2017,25(9):953-956.

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History
  • Received:May 05,2017
  • Revised:May 28,2017
  • Online: September 29,2017
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