激活巨噬细胞抑制载脂蛋白AI促细胞胆固醇流出能力
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Effect of Activation on the Capacity of Apolipoprotein AI to Promote Cellular Cholesterol Efflux in Murine Macrophages
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    摘要:

    为探讨巨噬细胞活化对载脂蛋白AI促胆固醇外流能力的影响,用炎症诱发剂活化鼠腹腔巨噬细胞,3h标记乙酰低密度脂蛋白作泡沫细胞诱导物,巨噬细胞衍化的泡沫细胞与载脂蛋白AI孵育一段时间后,测定由细胞释放入培养基中的3h标记胆固醇量。结果发现,载脂蛋白AI浓度为20mg/L时,从被活化的细胞释放至基质中的3h标记胆固醇量[(4.15±0.41)×10-1Dpm/g细胞蛋白]显著低于未被活化的细胞[(5.69±0.12)×10-1dpm/g细胞蛋白](p<0.01),但高密度脂蛋白清除细胞胆固醇能力两组细胞无显著差异。提示巨噬细胞活化可显著抑制载脂蛋白AI促细胞胆固醇外流能力。

    Abstract:

    Aim To investigate the effects of macrophage activation on the capacity of apolipoprotein AI to promote cellular cholesterol efflux. Methods 3H labled acetyl low density lipoprotein (ac LDL) was employed as an inducer for foam cell formation by thioglycolate were cultured with apo AI for 18 h, 3 H cholesterol released from cells to the medium was measured by scintillation spectrometry. Results The efflux of cholesterol from inflammatory macrophages to apo AI did not increase markedly with the increase of the concentration of apo AI as that observed in resident macrophages. The macrophage activation resulted in a significantly decrease in the capacity of apo AI (20 mg/L) to promote cellular cholesterol efflux (P<0.01), but had no effect on HDL. Conclusion The macrophages activation may effectively inhibit the capacity of apo AI to remove cholesterol from cells.

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张春妮,田秀,庄一义,堀内正公.激活巨噬细胞抑制载脂蛋白AI促细胞胆固醇流出能力[J].中国动脉硬化杂志,1999,7(1):29~31.

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