胆固醇诱导血管内皮细胞基因的差异表达
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国家重点项目研究发展规划(G2000057007)资助


Differentially Expressed Genes in Vascular Endothelial Cell Induced by Cholesterol
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    摘要:

    为探讨高胆固醇致动脉粥样硬化的分子机理,利用抑制消减杂交技术克隆胆固醇诱导脐静脉内皮细胞产生的差异表达基因,并对其表达进行初步研究。经顺、反两向消减杂交和巢式聚合酶链反应扩增,获得了差异表达的cDNA片段,克隆化后挑选部分进行测序、同源性比较及应用半定量逆转录—聚合酶链反应分析部分差异基因的表达情况,得到2 3个差异表达的cDNA片段,其中6个新差异表达cDNA片段被GenBank接受。已知基因中的血小板反应蛋白1和蛋白酶体亚单位基因表达的改变可能与高胆固醇致动脉粥样硬化作用相关

    Abstract:

    Aim To investigate cholesterol-induced atherogenesis molecular mechanism, clone and analyse the differentially expressed genes in vascular endothelial cell induced by cholesterol. Methods Suppression subtractive hybridization (SSH) method is used to isolate the differentially expressed cDNA in human umbilical vein endothelial cell induced by cholesterol. After sequencing and homology analysis, ten differentially expressed cDNA fragments are selected and analysed by reverse transcription-polymerase chain reaction(RT-PCR). Results Twenty-three differentially expressed cDNA fragments have been isolated. Seventeen of them are known genes, the other six are unknown genes. The known genes include thrombospondin-1, proteasome subunit (type and so on). The ten elective cDNA fragments (including four known genes and six unknown genes) that are identified by RT-PCR have the same expressed tendency as SSH discovery. Six differentially expressed unknown cDNA fragments have been accepted by GenBank as novel expressed sequence-tags (EST). Conclusion The high level expression of thrombospondin-1 and the low level expression of proteasome subunit (type) in endothelial cell induced by cholesterol may be related to cholesterol-induced atherogenesis.

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唐蔚青,王抒,杨向东,陈保生,李凌松.胆固醇诱导血管内皮细胞基因的差异表达[J].中国动脉硬化杂志,2003,11(1):1~4.

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  • 收稿日期:2002-08-12
  • 最后修改日期:2003-01-08
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