蜂胶抗动脉粥样硬化研究进展
DOI:
作者:
作者单位:

作者简介:

通讯作者:

基金项目:


Effects of Heat Shock Pretreatment on the Lipoplysaccharid-Induced Macrophage Migration
Author:
Affiliation:

Fund Project:

  • 摘要
  • |
  • 图/表
  • |
  • 访问统计
  • |
  • 参考文献
  • |
  • 相似文献
  • |
  • 引证文献
  • |
  • 资源附件
    摘要:

    动脉粥样硬化(As)是严重危害人类健康的常见病和多发病,其发生机制涉及脂质代谢障碍、氧化应激、血管内皮细胞损伤和平滑肌细胞迁移增殖、巨噬细胞浸润以及泡沫细胞形成和细胞坏死、凋亡等许多过程,因此针对其发病机制寻找药理作用广泛且无毒副作用的抗As药物或辅助药物无疑具有重要意义。蜂胶被誉为“紫色黄金”,是蜜蜂从植物芽苞和树干处采集的树脂,并混入蜜蜂上颚腺分泌物和蜂蜡而形成的一种具有芳香气味的胶状物,含有黄酮类....

    Abstract:

    Aim To study the effects of heat shock pretreatment on the lipopolysaccharid-induced macrophage migration. Methods The effect of lipopolysaccharid on the release of tumor necrosis factor-α(TNF-α) and interleukin-1β(IL-1β) in murine macrophage-like RAW264.7 cells were detected by ELISA;the effect of lipopolysaccharid on the migration of RAW264.7 macrophages were detected by chemotaxis assay;the effect of heat shock pretreatment on the migration of RAW264.7 macrophages were detected by chemotaxis assay. Results Interfered with 500 μg/L lipopolysaccharid for 1 h,the release of TNF-α and IL-1β were significantly increased(P<0.05) in RAW264.7 macrophages;interfered with 500 μg/L lipopolysaccharid for 24 h,the migration of RAW264.7 macrophages was significantly increased(P<0.05);given a heat shock pretreatment about 42.5℃ for 1 h followed with the interference of 500 μg/L lipopolysaccharid,the migration of RAW264.7 macrophages induced by lipopolysaccharid was significantly inhibited(P<0.05). Conclusion Heat shock pretreatment significantly inhibited lipopolysaccharid-induced macrophage migration.

    参考文献
    相似文献
    引证文献
引用本文

王家富,姚树桐,秦树存.蜂胶抗动脉粥样硬化研究进展[J].中国动脉硬化杂志,2009,17(7):620~621.

复制
分享
文章指标
  • 点击次数:
  • 下载次数:
历史
  • 收稿日期:2008-11-27
  • 最后修改日期:2009-05-31
  • 录用日期:
  • 在线发布日期: